In the Schacter and Singer study, subjects in the “euphoric model” (EM) condition received *exactly the same endogenous neurochemicals * as those in the “angry model” (AM) condition. Thus, it is highly unlikely that subjects in the EM condition would just by chance happen to have had more of the Hu et al “happiness” endogenous neurochemicals in their system than those in the AM condition and that those in the AM condition would just by chance happen to have had more of the Hu et al “anger” endogenous neurochemicals in their system than those in the EM condition. But that is what would have to have happened if Hu et al are right about the differnet endogenous neurochemicals being the cause of the differnet emotions.
In the unlikely event that such a confound exsted in the Schacter/ Singer study, making it all the more likely that Hu et al are correct about differnet endogenous neurochemicals being the cause of the different emotions reported by the subjects in the Schacter/Singer study, that would definitely put a nail in the coffin of the PCT nodel of emotion.